Here you go. "Remember that fructose does not stimulate insulin secretion. The hormonal steering of metabolism applies only to that induced by glucose. The excess substrate from fructose is poured into fat synthesis which is stimulated by the insulin released in response to simultaneously consumed glucose. The liver has a huge capacity for the uptake and phosphorylation of fructose. The phosphorylation capability is about twice that of the glucose phosphorylation system. We find large increases in uptake and metabolism of fructose with increased sugar levels in portal blood. In individuals with fructose intolerance we find an increase in fructose-1-phosphate due to the fact that fructokinase activity can exceed that of aldolase B. This special aldolase is required to cleave F-1-P to dihydroxyacetone phosphate and glyceraldehyde. The fructose taken up cannot be stored as such. It is converted to intermediates in glycolysis WHICH COME AFTER THAT ALL-IMPORTANT CONTROL POINT, PHOSPHOFRUCTOKINASE 1. So what? Well, some of that fructose winds up as glycogen and glucose. That is ok; we need glucose in the blood. The rest of the fructose rushes through pyruvate, is transferred to mitochondria, and finally converted to fatty acids and exported from the liver as triglycerides.