I think you're right, but the issue for me has been not cutting back the Metformin to the point my fasting BGs are above 120 mg/dL, while maintaining everything else. Unfortunately (or fortunately, depending on how you look at it), my only two tools for changing my BG are eating (or not eating) and taking Metformin (or not taking it). Balancing carb intake with exercise and Metformin dosage to maintain weight while not having high BG has been... challenging for me.
And I am very fortunate, and understand that. I have the easiest damn case of Type 1 on the planet I'm pretty sure, at least for now. The only thing that bothered me about the Endo's directions were the advice to raise my A1c. She was absolutely convinced that, as a diabetic, I should not have a "normal A1c or blood glucose." The logic seems tortured to me, since I'm not on insulin: it is commonly assumed that low A1cs (below 5.5%) are derived from frequent hypo events caused by insulin reactions. However, in my case I have very infrequent and mild lows (like...two or three a quarter) resulting from intense endurance exercise (or work). My A1c isn't a reflection of frequent insulin-caused lows or the infrequent exercise lows I actually have, it's a reflection of that I'm not overloading my endogenous insulin production with more carbs than I can handle and my use of a low-dose Metformin.
If I was taking exogenous insulin and experiencing any lows at all, I could totally understand the direction to relax the strict control. It seems completely nonsensical to me, on the other hand, to purposely compromise my fasting BG and postprandial BG in order to have an A1c between 5.5% and 6.0% (which she believes would be "ideal" for me as a diabetic). I'm not sure she's wrong, but I'm having difficulty figuring out why I should run higher than "normal" BGs just because I'm diabetic when I'm otherwise non-symptomatic (at least with my medication and diet). The other bit of "iffy" direction I got is that when my BG does become compromised, she said she'd prefer I add another oral rather than insulin.
For whatever reason, I seem to live in an "insulin as a last resort" corner of the U.S. I'm serious about that: despite being antibody positive, I've had three doctors (two primary care and one endo) state that the goal of my management should be avoiding insulin dependency as long as possible. All of them describe acute hypos as the real danger of being Type 1, and all of them view complications of high BG as being unavoidable. I find that...incompatible with what I've learned from Type 1s here and elsewhere. When I questioned my first doctor about this, he gave me a story about how he lost a med-school professor who died from a critical low, and this was supposed to be taken as a cautionary tale on why I should never (if possible) use insulin.
Anyhow, I couldn't possible use insulin now other than as an occasional correction for a postprandial or the rare persistent, mild high BGs I have every once and awhile. So, the "treat this as if Type 2 and eat to fuel exercise" direction makes perfect sense, for now. Not sure I'm going to purposely raise my A1c, but I will definitely try to start training again with eating to up my BG beforehand.
As for orange juice...I used to be a drunk, and I drank far too many screwdrivers. Even the smell of OJ makes me sick to my stomach these days, and I can't imagine that being the only cure for a low! But yes, even in my limited experience (of having to eat my way out of a persistent low BG from exercise), I really dislike it when I have to eat. I recently tried to gain some weight through heavy lifting and eating like I was 16 again, and I more or less gave up because I literally couldn't force myself to eat 4,000 calories a day (and wasn't gaining weight anyways).