Comment

Comment by sarhow on June 3, 2010 at 5:05am

I've written a guest blog, "Can Environmental Contaminants Contribute to the Development of Diabetes?" It is online at http://ourhealthandenvironment.wordpress.com/

Comment by sarhow on May 23, 2010 at 4:41am

There's a new study out (free full text) that used a computer model to analyze diabetes and 266 various environmental factors:

http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone....

The database used doesn't distinguish between type 1 and type 2, but most people would have type 2. It found that PCBs and a pesticide were most strongly associated with diabetes. These environmental factors were as important as genetic factors found in genome-wide studies.

Comment by Zolar1 on May 20, 2010 at 9:09pm

Please research 'xenoestrogens' and estrogen dominance.

Comment by D.D. on May 20, 2010 at 8:31am

I started a discussion with the members of this group. I am very interested in science and exploring these topics and questions with like-minded people. Thanks for starting this group. DD

Comment by sarhow on April 12, 2010 at 11:24am

Why are more and more children diagnosed with type 1 diabetes every year?

The website www.diabetesandenvironment.org summarizes the peer-reviewed, scientific evidence relating to the various factors that may increase or decrease the risk of type 1 diabetes. While some of these factors may look familiar, such as viruses, cow's milk, or vitamin D deficiency, others are not as well known, namely environmental toxins. Some toxins have been found to affect the beta cells that produce insulin in the lab ... some toxins have been found to initiate or worsen autoimmunity.

There is growing evidence that environmental contaminants may also contribute to the development of type 2 diabetes, and this evidence is included on the website as well. A provocative study of U.S. residents found that people who were obese but had very low levels of contaminants in their bodies did not have increased rates of diabetes. Only in people with certain levels of contaminants was obesity associated with diabetes. Not only that, but exposure to some contaminants may also lead to weight gain. In other words, obesity alone may not be the whole story. We may need to rethink our preconceptions about diabetes altogether.

Do environmental contaminants contribute to the rising incidence of both type 1 and type 2 diabetes? Can preventing pollution also prevent diabetes?

I think these questions are worth asking. I would like to meet others who are also interested in this possibility.

Sarah

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