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This seems to be a burning topic that I thought I’d answered in my previous post but because of Halle Berry
and YouTube videos like “Raw for thirty days”, I must revisit the topic.
What does it mean to “wean” anybody from insulin? It means to withdraw it from their system, take it away from them. People, none of us can live without insulin! The body isn’t accepting any substitutes, either. Those who have been “weaned” from taking insulin obviously have working beta
cells which produce insulin. If they did not, it would probably be over pretty quickly.
The claim for this “weaning” then is based on the reanimation of dead beta cells through various techniques. Now what makes this plausible are the testimonies of many individuals who were diagnosed as T1 and then found themselves able, through the use of (name your technique), to become insulin independent. These people are adamant. They were DKA, might have even
gone into a coma. They were thoroughly dependent upon insulin injections, for maybe years and now they don’t need it.
Unlike some, I’m not going to call them liars, I will even vouch for their experience but I want to examine this rationally.
The very first thing to note is that they were diagnosed and told they were T1 because of severe hyperglycemia, DKA, weight loss and maybe or maybe not the presence of antibodies. They were given the ADA diet of 200 to 300 grams of carbs a day, with most of the carbs coming from healthy grains and stuff like that.
They believed the diagnosis, as anyone would, but they, just like most of us experimented with their situations. The major thing that they all found was “Bernsteins’ law of small numbers”. If carbs are reduced then the insulin adjustment can be reduced as well. This simply means a change in
diet. If the carbs are going down something else must be replacing them. If you now throw in some sort of physical regimen then you’ve got something going. This is a type of “weaning” but reducing the insulin need isn’t the same as producing insulin. How does that happen? It doesn’t. You either have it or you don’t. But a person might object saying that they didn’t produce hardly any insulin before and now they don’t need it and this has gone on longer than any honeymoon on record.
The key to this is diagnosis. At the point of diagnosis, they were given the T1 designation, because there isn’t another. What if there were? Well, there is, the problem is there is no way to distinguish a T1 from a Ketosis Prone T2 Diabetic at diagnosis. It is only after months and with a c-peptide test can they be told apart. This is because, for whatever reason, a KP T2 can regenerate their beta
cells once glucose levels return to normal even in the face of testing positive for antibodies.
The other key is the ADA diet. This diet is high in carbohydrates and KPD’s have a 20% higher deficiency in G6PD then even regular T2 diabetics. This is a significant disadvantage in handling carbs. High carb intake will keep them from regenerating beta cells and they will effectively stay T1. A KPD T2 is the only known diabetic that can go from a T1 status (no beta cell functioning, no insulin) to a T2 status.